MINISTRY OF HEALTH -------	SOCIALIST REPUBLIC OF VIETNAM Independence - Freedom - Happiness --------------
No.: 167-BYT/QD	Hanoi, February 04, 1997

 

DECISION

ON ADDITIONAL ISSUANCE OF OCCUPATIONAL DISEASES IN THE LIST OF COVERED OCCUPATIONAL DISEASES

MINISTER OF HEALTH

Pursuant to Article 106 of the Labor Code on the issuance of the list of occupational diseases;

Pursuant to Decree No. 68 dated October 11,1993 of the Government stipulating functions, duties and powers of the organizational structure of the Ministry of Health;

Pursuant to official dispatch No. 334/LDTBXH-BHLD dated January 29, 1997 of the Ministry of Labour - Invalids and Social Affairs authorizing the Ministry of Health to sign the decision on addition of 5 occupational diseases to the list of covered occupational diseases 1997;

After obtaining the consent of Vietnam General Federation of Labor in the official dispatch No.1592/TLD dated December 31, 1996;

DECIDES

Article 1. Additional issuance of 5 occupational diseases to the list of covered occupational diseases (attached annexes)

1. Occupational arsenic and arsenic compound intoxication disease (Annex 1).

2. Occupational nicotine intoxication disease (Annex 2).

3. Occupational pesticide intoxication disease (Annex 3).

4. Occupational caisson disease (Annex 4).

5. Occupational chronic bronchitis (Annex 5).

Article 2. The laborers who are inspected to suffer from diseases specified in Article 1 of this Decision shall be entitled to the benefits specified in the Labor Code and documents guiding the implementation of this Labor Code.

Article 3. This Decision takes effect from its signing date.

Article 4. Chief of Office, Director of Hygiene and Disease Prevention Department and Departments under the Ministry of Health, Head of units directly under the Ministry of Health, Director of Service of Health of provinces and centrally-affiliated cities, head of health sector and heads of relevant units are liable to execute this Decision.

 

Nguyen Van Thuong (Signed)

 

ANNEX 1

OCCUPATIONAL ARSENIC AND ARSENIC COMPOUND INTOXICATION DISEASE
(Issued together with Decision No. 167 dated February 04, 1997)

I. MAIN OCCUPATIONS AND JOBS MAY CAUSE DISEASE

All jobs with exposure to or breath of arsenic dust or vapor and arsenic compounds.

- Arsenic ore processing.

- Production and use of arsenic chemical pesticides.

- Ore processing in arsenic non-ferrous metallurgy

- Use of arsenic compounds and inorganic substance in leather processing, production of glass, electronic parts…

II. DIAGNOSIS INSTRUCTION:

1. Diagnosed subject:

Diagnosed subject are laborers working in the environment with arsenic dust, vapor or arsenic inorganic compounds.

2. Exposure time:

Determined by the concentration of exposure and disease;

. Acute disease: typically short duration of exposure with high concentrations.

. Chronic disease: with concentration lower than the permit standard but long duration of exposure may also be affected.

3. Sub-clinical signs:

Urinary arsenic amount: The arsenic amount in urine must be greater than or equal to 100 † g / l (or †g creatinine) (it is necessary to get the urine for every 24 hours).

Eating fish and sea food shall increase the urinary arsenic amount. Thus, the subjects diagnosed must avoid eating sea food at least for 2 days before collecting urine for determination of arsenic amount .

4. Clinical signs and symptoms:

4.1. Acute intoxication:

- Nausea and diarrhea

- Severe abdominal pain

- Little urination

- Reduction in body temperature and blood pressure

- Cramp and convulsion.

For acute intoxication due to AsH3, there are signs as follows:

- Urination with hemoglobin

- Jaundice and thrombolysis

- Plasma proteins in nephritis

- Central nervous system toxicity (coma)

4.2. Chronic intoxication:

. Initial symptoms: irritability, abdominal pain, itching, pain in joints and asthenia.

. Signs: Diarrhea or constipation, rash, gauntness, swelling of lower eyelid, mucosal lesion, gingivitis, pharyngitis, upper respiratory inflammation (runny nose, hoarseness, cough ...) and combined membrane inflammation.

. Neurological symptoms: Feeling of numbness, skin burning, tingling or itching accompanied with tremor, muscle twitching, muscle atrophy, limb paralysis. Inflammation of many nerves is a primary sign.

. Skin and mucous membrane lesion: Inflammation, ulcer, palmoplantar keratodermas

. Skin darkening and hair loss

. Hepatic impairment

. Nephritis and kidney failure

4.3. Cancer: Cancer of the skin, lungs, ethmoid bone, malignant warts.

III. INSPECTION INSTRUCTION

Lesion – Sequelae after treatment	Guarantee time	Rate of loss of working capacity (%)	Note
I. Arsen and arsenic organic compounds
1. Skin and mucous membrane lesion:
a. Contact dermatitis causing chronic ulcer	1 month	5-10	Still recurring after over 03 times of treatment
If more than one ulcer (over 3mm and more than 05 ulcers)		11-15
b. Skin ulcer successfully treated, stable scar	As above	1-5
c. Perforation of the nasal septum	As above	11-15
d. Chronic conjunctivitis and blepharitis	As above	11-15
Keratitis with remaining scar affecting vision			Assessed by central vision table (Table 1)
e. Skin darkening (depending on region and area):	3 months
- Less than 50% of face and neck area		11-15
- More than 50% of face and neck area		16-20
- Less than 50% of arm and leg area		6-10
- More than 50% of arm and leg area		11-15
g. Palmoplantar keratodermas	as above	5-10
2. Neuromuscular lesion	6 months
a Neuromuscular dermatitis affecting movement
- Mild degree (less affecting movement)		21-25
- Moderate degree (Movement with difficulty)		26-31	³2 limbs =31%
- Heavy degree (Movement with extreme difficulty)		31-40	³2 limbs =41%
b. Flaccid paralysis and muscular atrophy:	6 months
* With limbs:
. Mild degree (working with limitation)		21-25
. Moderate degree (working with difficulty)		35-40	2 limbs =41%
. Heavy degree (loss of working capacity)		61-65
* Flaccid paralysis and muscular atrophy not in limbs		16-20
c. Sequelae of lesion to the central nerve			Rating by Sequelae VII of neuropathy group of classification standard of loss of working capacity (Table 2)
3. Forms of cancer due to arsen:	30 years
- Primary carcinoma		61-65
- Liver sarcoma		81-95
- Primary lung cancer		81-95
II. Asen hydro hay Arsin (AsH3) Arsenic hydrogen or Arsin (AsH3)	30 days
1. Jaundice and thrombolysis after acute intoxication
a. Erythrocyte E 3 T. HST E 11 g%		31-35
b. Erythrocyte E 2.5 T. HST E 10 g%		41-45	Temporarily classified and re-inspected after 01 year
2 Hepatic impairment
a. Liver function changed little (test after treatment)		31-35
b. Liver function changed much		41-45	Temporarily classified and re-inspected after 01 year
3. Plasma proteins in nephritis	60 days
a. Blood urea E 0.6 g / l		31-35
b. Regular blood urea from 0.6 to 1 g / l		41-45
c. Regular blood urea ³ 1 g/l		51-55
4. Irreversible chronic renal failure (ascites, HC <2,000,000 blood urea> 1.5 g / l creatinine> 100 mmol / 1)		61-70
If there are serious complications such as paralysis and blindness		81-85

Note:

When being intoxicated with arsen and acute arsenic compounds, first aid and treatment must be performed until stable, if any new Sequelae occurs, it should be inspected by the Medical Evaluation Board.

. When the disease recurs, it must be treated until stable and then re-inspected.

Persons suffering from diseases mentioned above (defined in recruitment dossier) shall not have medical inspection to determine occupational diseases.

 

ANNEX 2

OCCUPATIONAL NICOTINE INTOXICATION DISEASE
(Issued together with Decision No. 167 dated February 04, 1997)

I. MAIN OCCUPATIONS AND JOBS MAY CAUSE DISEASE

All jobs with exposure to or breath of cigarette and nicotine dust such as:

. The work during the process of cigarette production: tobacco stemming, drying, screening, material coating, julienne, cigarette rolling, bagging ...

. Tobacco harvesting, packing, shipping ...

II. DIAGNOSIS INSTRUCTION:

1. Diagnosed subjects: are persons working in environment with cigarette and nicotine dust.

2. Exposure time:

Determined by the concentration of exposure and disease;

. Acute disease: typically short duration of exposure with high concentrations.

. Chronic disease: with concentration lower than the permit standard but long duration of exposure may also be affected.

3. Sub-clinical signs:

Urinary nicotine amount:

- For non-smokers: the urinary nicotine amount is more than 0.3 mg/l.

- For smokers: the urinary nicotine amount is more than 0.3 mg/l.

4. Clinical signs and symptoms:

4.1. Acute intoxication:

- Severe dizziness and headache, pale face.

- Nausea, vomiting, abdominal pain and diarrhea.

- Saliva secretion and cold sweat streaming

- Rapid heartbeat, increased blood pressure, pain in the heart

- Visual and hearing disturbances

- Eyelid vibration, hand tremor and muscle cramp.

4.2. Chronic intoxication:

- Mucosa: with phenomenon of irritability, dry gular and nasal mucosa, stomatitis, conjunctivitis (watery eyes, eye pain, vision loss).

- Skin and nails: atopic dermatitis, thin and brittle nails;

- Cardiovascular: heart attack, heartbeat change, premature ventricular beat, blood pressure change.

- Nerve: headache, poor sleep, decreased memory, easy to forget, decreased hearing and vision, tremor.

- Digestion: nausea, loss of appetite, indigestion, diarrhea, heartburn, epigastric pain.

- Respiration: chronic bronchitis, alveolar expansion, decreased pulmonary ventilation.

III. INSPECTION INSTRUCTION

Lesion – Sequelae after treatment	Guarantee time	Rate of loss of working capacity (%)	Note
1. Skin and mucosa	3 months
a. Chronic conjunctivitis		5-10
b. Dermatitis, chronic exposed skin due to allergia.
- Inflamed area ³ 20% of parts (limbs, face, neck…)		21-25
- Inflamed area E 20%		10-20
2. Circulatory apparatus dysfunction	3 months
a. Hypotension (systolic blood pressure E 90 mmHg; diastolic blood pressure E 60 mmHg)		16-20	Table 3
b. Blood pressure decreases > 160/90 mmHg, stage 1-2		16-20	-as above-
c. Premature ventricular arrhythmias
- Infrequent ³ 12 beats / min		10-15
- Fast (antiarrhythmic drugs must be used regularly)		25-30
d. Slow beat (less than 55 times / min)		21-25
- There is atrioventricular block grade 3 but without faint		35-40
- There is atrioventricular block grade 3 with faint, successful treatment		45-50
- There is atrioventricular block grade 3 with faint, unsuccessful treatment		61-70	Temporary rating,re-inspection after 1 year
e. Lesions to the coronary arteries and heart muscle		35-40
3. Neurasthenia syndrome	3 months	25-30
(Headache, fatigue, sleeping difficulties, memory loss to be under prolonged treatment over a year)

Note:

. When being intoxicated with nicotine, first aid and treatment must be performed until stable, if any new Sequelae occurs, it should be inspected by the Medical Evaluation Board.

When the disease recurs, it must be stably treated and then re-inspected.

 Persons suffering from diseases mentioned above (defined in recruitment dossier) shall not have medical inspection to determine occupational diseases.

 

ANNEX 3

OCCUPATIONAL PESTICIDE INTOXICATION DISEASE
(organophosphate, organochlorine, carbamate)
(Issued together with Decision No. 167 dated February 04, 1997)

I. MAIN OCCUPATIONS AND JOBS MAY CAUSE DISEASE

All jobs with exposure to chemical pesticdes:

- Industrial production.

- Packaging

- Transport

- Storage

- Mingling, mixing, spraying, sprinkling, steaming ...

II. DIAGNOSIS INSTRUCTION

1. Diagnosed subjects:

Diagnosed subjects: are persons who are exposed to chemical pesticides.

2. Exposure time:

Determined by the concentration of exposure and disease;

. Acute disease: typically short duration of exposure with high concentrations.

. Chronic disease: with concentration lower than the permit standard but long duration of exposure may also be affected.

3. Sub-clinical signs:

Amount of Acetylcholinesteraza enzyme activity (AChE): The amount of AChE enzyme activity decreases over 25% compared to that before exposure or the constant of AChE enzyme activity in normal person.

4. Clinical signs and symptoms:

4.1. Acute intoxication:

- Vomiting and abdominal pain

- Sweating, watery eye, runny nose, saliva secretion…

- Miosis

- Pulmonary edema

- Muscle twitching and cramping

- Paralysis, coma.

4.2. Chronic intoxication:

- Headache, dizziness

- Fatigue

- Poor sleep

- Loss of appetite

- Hand tremor

- Eyeball twitching

- Light paralysis

- Skin manifestations: rash, eczema ...

- Neurovegetative dysfunction

III. INSPECTION INSTRUCTION

Lesion – Sequelae after treatment	Guarantee time	Rate of loss of working capacity (%)	Note
1. Skin: dermatitis and contact eczema	30 days	5-10
2. Neurological sequelae	90 days
a. Nystagmus may affect vision
- One eye		5-10
- Two eyes		11-15
b. Local muscle quivering		5-10
c. Paralysis (depending on paralyzed muscular group, in one or more limbs, in any area of the body and degree of paralysis)
- Mild degree (working with limitation)		21-25
- Moderate degree (working with difficulty)		35-40
- Heavy degree (paralyzing entire limb and loss of working capacity)		61-65
d. Neurasthenia syndrome and neurovegetative dysfunction	9 days	25-30
3. Chronical intoxication of chemical pesticide with organochlorine	180 days
a. Hepatic impairment, decreased liver function lasting months
- Mild degree		31-35
- Moderate and severe degree		45-58
- If progressing, thus becoming cirrhosis ascites		61-70
b. Chronic nephritis and increased blood urea			Determination of renal tubular lesion, rate of loss of working capacity according to the similar plasma urea toxicated with AsH3
c. Anemia marrow (After poisoning of Chlordan and Lindan)
- HC 3,000,000, HST E11 g%		31-35
- HC E 2,500,000, HST E 10 g%		41-45
- HC E 2,000,000, HST E 8 g%		61-65

Note:

When being intoxicated with chemical pesticide, first aid and treatment must be performed until stable, if any new sequela occurs, it should be inspected by the Medical Evaluation Board.

When the disease recurs, it must be stably treated and then re-inspected.

Persons suffering from diseases mentioned above (defined in recruitment dossier) shall not have medical inspection to determine occupational diseases.

 

ANNEX 4

OCCUPATIONAL CAISSON DISEASE
(Issued together with Decision No. 167 dated February 04, 1997)

I. MAIN OCCUPATIONS AND JOBS MAY CAUSE DISEASE

All jobs carried out under conditions of pressure high than atmospheric pressure: divers, working in the sank cage ...

II. DIAGNOSIS INSTRUCTION:

1. Diagnosed subjects:

Diagnosed subjects are persons working in conditions of high pressure or compressed air (divers and person working in sank cage).

2. Exposure time:

Exposure time may vary with disease: acute or chronic.

. Time of apprearance of acute disease may be right after pressure reduction.

. For chronic disease, the exposure time is usually 01 year.

3. Clinical signs and symptoms:

3.1. Acute caisson disease:

- Pain in limbs

- Vomiting, epigastric pain

- Dizziness

- Tingling and numbness in limb tip

- Shortness of breath

- Headache

- Epilepsy

- Visual disturbances, dazzling or scotoma

- Heart attacks, coronary artery disorder, irregular heartbeat.

- Hypotension

3.2. Chronic caisson disease:

- Limb fatigue and pain

- Difficult movement: stiffness, less or much limitation of movement.

- Muscle atrophy

- Hearing loss.

4. Sub-clinical signs (chronic decompression)

4.1. X-ray signs: detection of bone changes:

- Calcium disorders: mineral loss

- Bone structure: bone cavity

- Dissolution of bone

- Periosteal reaction (spine, thick bone)

Change of bone found in the limb root: shoulders, groin. As seen in the upper and lower end of femur, tibia, in the upper end and shaf of humerus bone.

4.2. Audiometry: to identify hearing loss

III. INSPECTION INSTRUCTION

Lesion – Sequelae after treatment	Guarantee time	Rate of loss of working capacity (%)	Note
1. Vestibular syndrome (dizziness, loss of balance) determined by experimental method of labyrinth	3 months
- Mild degree		15-20
- Moderate degree (working with limitation)		31-35
- Heavy degree (working with difficulty)		45-50
2. Chronic inflammation of the middle ear, perforated eardrum	3 months
- One ear		10-15
- Two ear		25-31
3. Occupational hearing loss, with or without labyrinth disorder, no progress after stopping work in high pressure, determined by complete audiometry from 3-6 months after stopping work in high pressure .	12 months
- Mild hearing loss of both ears		15-20
- Moderate hearing loss of both ears		26-31
- Heavy hearing loss of both ears		41-51
- Totally deaf of both ears		61-70	Table 5
4. Local myocardial ischemia
a. Infrequent and mild heart attacks (type 2 according to NYHA)		35-40	Table 3
b. Fast heart attacks affecting activities (type 3 according to NYHA)		51-60	-as above-
c. Enlarged heart, heart failure, with old myocardial infarction.		71-80
5. Osteonecrosis  (determined by X-ray)	20 years
- In 01 joint or 1 bone		21-30
- In 01 joint or 2 bones		31-40
- More than 02 joints or 2 bones		45-60
6. Paralysis of limbs	3 months		Rating by Sequelae VII of neuropathy group of classification standard of loss of working capacity (Table 2)

Note:

The working complications in high-pressure must have first aid and treatment until stable, if any new Sequelae occurs, it should be inspected by the Medical Evaluation Board.

When the disease recurs, it must be stably treated and then re-inspected.

Persons suffering from diseases mentioned above (defined in recruitment dossier) shall not have medical inspection to determine occupational diseases.

 

ANNEX 5

OCCUPATIONAL CHRONIC BRONCHITIS
(Issued together with Decision No. 167 dated February 04, 1997)

I. MAIN OCCUPATIONS AND JOBS MAY CAUSE DISEASE

All jobs with exposure to organic and inorganic dust or toxic gas such as CO, SO2, H2S, C1, HCL etc. ..

II. DIAGNOSIS INSTRUCTION:

1. Diagnosed subjects

Diagnosed subjects are persons who are exposed to some kinds of dust or toxic gas such as SO2, H2S etc. ..

2. Exposure time: The prescribed exposure time is 3 years.

3. Clinical signs and symptoms:

Coughing and spitting sputum for over 2 months in a year and over 2 consecutive years.

4. Sub-clinical signs:

Measurement of respiratory function: maximum expiratory volume / second (FEV1) declines.

III. INSPECTION INSTRUCTION

Lesion – Sequelae after treatment	Guarantee time	Rate of loss of working capacity (%)	Note
1. Common chronic bronchitis does not affect heart	12 months
Irreversible decline in FEV1:
- Degree I		15-20
- DegreeII		21-30
- Degree III		31-40
2. Chronic bronchitis with spasm and allergy	12 months
Irreversible decline in FEV1:
- Degree I		31-35
- DegreeII		41-45
- Degree III		51-55
3. Chronic bronchitis with respiratory failure and heart failure (right sided heart failure)
- Stage I		41-50
- Stage II		51-60
- Stage III		61-75
- Stage VI		81-90

Note:

The persons who are given inspection of occupational chronic bronchitis must have the result of measurement of respiratory function in normal physiological limit at least 3 years before

 

TABLE 1

TABLE OF PERCENTAGE OF LOSS OF WORKING CAPACITY DUE TO VISION LOSS BECAUSE OF LESION OF VISUAL ORGAN

Vision	9/10 8/10	7/10 6/10	5/10	4/10	3/10	2/10	1/10	1/20	Below 1/20	ST (-)
9/10 8/10	0	5	8	11	14	17	21 20	25	31 30	41
7/10 6/10	5	8	11	14	17	21 20	25	31 30	35	45
5/10	8	11	14	17	21 20	25	31 30	35	41 40	51 50
4/10	11	14	17	21 20	25	31 30	35	41 40	45	55
3/10	14	17	21 20	25	31 30	35	41 40	45	51 50	61
2/10	17	21 20	25	31 30	35	41 40	45	51 50	55	65
1/10	21 20	25	31 30	35	41 40	45	51 50	55	61 60	71 70
1/20	25	31 30	35	41 40	45	51 50	55	61 60	71 70	81
Below 1/20	31 30	35	41 40	45	51 50	55	61 60	71 70	81 80	85
ST (-)	41	45	51 50	55	61	65	71 70	81	85	91

 

TABLE 2

VI. GROUP OF NEUROPATHY

No.	Name of disease – Type of disease	Percentage of loss of working capacity (%)
(1)	(2)	(3)
1	Sequelae of hemiplegia or simply two legs (from any cause whatever)
	a. Sequelae of hemiplegia:
	- Mild degree: almost normal movement	41-45
	- Moderate degree: movement with difficulty
	If without teres disorder	55-60
	If with teres disorder	65-70
	- Heavy degree: unable to move (with or without teres disorder)	81-85
	b. Sequelae of paralysis of both legs
	- Mild degree: without teres disorder	50-55
	- Moderate degree: movement with difficulty, with or without teres disorder	65-70
	- Heavy degree	81-85
2	Sequelae of paralysis of one arm and one leg
	- Mild degree	30-35
	- Moderate degree	35-40
	- Heavy degree	55-60
3	Sequelae of lesion of one or many sensory nerves due to inflammation or pain
	- Mild degree	25-30
	- Moderate degree	35-40
	- Heavy degree	41-45
4	Cerebellar syndrome
	- Mild degree	25-30
	- Moderate degree	55-60
	- Heavy degree	61-65
	- Extremely heavy degree	81-85
5	Parkinson's syndrome
	. Mild degree: still able to work	41-45
	. Moderate degree: unable to work, still serve oneself	61-65
	. Heavy degree: unable to serve oneself	81-85
6	Lateral sclerosis, muscular dystrophy:
	- Mild degree	41-45
	- Moderate degree	61-65
	- Heavy degree	81
7	Other diseases and syndromes of the central nervous system
	* Migrain syndrome
	. Occurring 1-2 times/year	10-15
	. Occurring 1-2 times/ month	21-25
	. Weekly occurring affecting work	31-35
	* Local epilepsy
	. Occurring at times	20-25
	. 1-2 times/month	21-25
	. Regularly occurring	35-40
	* Total epilepsy
	. Rare crisis	25-30
	. Infrequent crisis	35-40
	. Fast crisis	61-65
	. Dementia (if any)	81-85
	* State equivalent to epilepsy or mental absence
	. Occurring 1-3 crises/year	10-15
	. Occurring 1-2 crises/month	25-30
	. Weekly occurring	35-40
	. Daily occurring	45-50
	* Mental epilepsy
	- Mild degree	40-45
	- Moderate degree	50-55
	- Heavy degree	70-75
	* Sweating limbs
	. Little effect on activity and working	21-25
	. Significant effect on activity and working	30-35

 

TABLE 3

CARDIOVASCULAR CLASSIFICATION ACCORDING TO NYHA
(NEW YORK HEART ASSOCIATION)
(Currently applied by the World Health Organization)

Type I: No symptom (shortness of breath, chest pain, palpitation) at rest or exertion.

Type II: No symptom (as above) at rest, but symptoms do appear upon performance of daily work.

Type III: No symptoms at rest, but symptoms do appear upon performance of a lighter job than the daily one.

Type IV: The symptoms do appear even at rest and only moving very slightly.

 

TABLE 4

CLASSIFICATION OF DEGREE OF HYPERTENSION DISEASE
(According to WHO)

I. CLASSIFICATION

Stage I:

Patient has no objective signs of any physical injury (see below).

Stage II:

Patient has at least one of signs of physical injury as follows:

+ Left ventricular hypertrophy can been seen upon clinical examination or electric ray, ECG, ultrasound cardiogram etc. ..

+ Widespread or localized narrow retinal arteries.

+ Proteinuria and / or serum creatinine increasing slightly.

Stage III:

Hypertension disease has caused injury to various organs, represented by the functional symptoms and physical signs as follows:

+ Heart: left ventricular failure.

+ In the brain: Hemorrhage of cerebrum, cerebellum or brainstem, hypertensive cecaphalopathy

+ Ocular fundus: retinal hemorrhages and exudation, with or without papilledema. These signs represent the progressive malignant stage.

Also in stage III hypertension or even other unclear signs which are the direct result of hypertension with these above signs, such as:

+ Heart: angina pectoris and myocardial infarction.

+ Brain: intracranial arterial thrombosis

+ Blood vessels: dissequant anevrysme (anevrysme dissequant) arterial inflammation.

+ Kidney: renal failure

II. SEVERITY CLASSIFICATION OF HYPERTENSION NUMBER

 (Measuring 03 times for every 02 weeks)

Classification	Systolic blood pressure (mm Hg)	Diastolic blood pressure (mm Hg)
1. Limited hypertension	140 - 159	90 - 94
2. Mild hypertension	160 - 189	95 - 104
3. Moderate hypertension	190 - 219	105 - 114
4. Heavy hypertension	220 or more	115 or more